Flucloxacillin and the HLA-B*5701 gene
Your individual risk of side effects in the event of treatment with flucloxacillin may be partly explained by genetic variations. It is known, for example, that mutations in the HLA-B*5701 gene may be partly responsible.
If you are HLA-B*5701 positive, the risk of severe liver damage is increased.
The absolute risk of liver damage remains extremely small: roughly 1 in 500 to 1,000 carriers will actually suffer liver damage.
Information about your genetic predisposition may provide grounds for extra vigilance in relation to a treatment with flucloxacillin.
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