If a member of your family is diagnosed with type 2 diabetes, you are naturally concerned, in the first instance, about the person affected by this disease. Later, however, other concerns will often also start to play on your mind. Is it hereditary? Will I get it too? Can I do anything to reduce the risk?
At iGene we deal with heredity and prevention on a daily basis and help you find answers to these kinds of questions.
Type 2 diabetes develops when the body can no longer effectively regulate your blood sugar. As too little insulin is present in the body, insufficient sugar can be removed from the blood. More and more young people are being affected by the condition. The majority of patients suffer from type 2 diabetes.
Genetic predisposition to type 2 diabetes
Your personal risk of type 2 diabetes depends on various factors. One of these is your DNA, but environmental factors and lifestyle also play an important role. To make a statement about your genetic predisposition, we examine mutations (changes or errors) in your DNA.
Preventing type 2 diabetes
Scientists are finding out more and more about ways to reduce the risk of type 2 diabetes. That means you can take steps yourself to prevent type 2 diabetes. iGene can help by sharing relevant scientific information with you in your iGene Passport.
Want to reduce your risk of type 2 diabetes?
An iGene Passport allows you to see straight away whether you have an increased or reduced risk of a large number of hereditary conditions, including type 2 diabetes. If it becomes apparent that you are at an increased risk of type 2 diabetes, you will also receive targeted information on how to reduce this risk.
A healthy lifestyle thanks to your iGene Passport
For as long as your iGene Passport is active you will continue to receive information based on the latest scientific insights. That means that if important discoveries are made in the future that will help you take even more effective steps to prevent type 2 diabetes, you will be able to benefit from them straight away.